Ilya Fleidervish
Ilya Fleidervish
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Slow inactivation of Na+ current and slow cumulative spike adaptation in mouse and guinea‐pig neocortical neurones in slices.
IA Fleidervish, A Friedman, MJ Gutnick
The Journal of physiology 493 (1), 83-97, 1996
Inward sodium current at resting potentials in single cardiac myocytes induced by the ischemic metabolite lysophosphatidylcholine.
AI Undrovinas, IA Fleidervish, JC Makielski
Circulation research 71 (5), 1231-1241, 1992
Na+ imaging reveals little difference in action potential–evoked Na+ influx between axon and soma
IA Fleidervish, N Lasser-Ross, MJ Gutnick, WN Ross
Nature neuroscience 13 (7), 852-860, 2010
Upregulation of KCC2 activity by zinc-mediated neurotransmission via the mZnR/GPR39 receptor
E Chorin, O Vinograd, I Fleidervish, D Gilad, S Herrmann, I Sekler, ...
Journal of Neuroscience 31 (36), 12916-12926, 2011
Kinetics of slow inactivation of persistent sodium current in layer V neurons of mouse neocortical slices
IA Fleidervish, MJ Gutnick
Journal of Neurophysiology 76 (3), 2125-2130, 1996
Persistent sodium current in layer 5 neocortical neurons is primarily generated in the proximal axon
N Astman, MJ Gutnick, IA Fleidervish
Journal of Neuroscience 26 (13), 3465-3473, 2006
Autoantibodies to the glutamate receptor kill neurons via activation of the receptor ion channel
M Levite, IA Fleidervish, A Schwarz, D Pelled, AH Futerman
Journal of autoimmunity 13 (1), 61-72, 1999
Functionally distinct NMDA receptors mediate horizontal connectivity within layer 4 of mouse barrel cortex
IA Fleidervish, AM Binshtok, MJ Gutnick
Neuron 21 (5), 1055-1065, 1998
Spatial mismatch between the Na+ flux and spike initiation in axon initial segment
G Baranauskas, Y David, IA Fleidervish
Proceedings of the National Academy of Sciences 110 (10), 4051-4056, 2013
Enhanced spontaneous transmitter release is the earliest consequence of neocortical hypoxia that can explain the disruption of normal circuit function
IA Fleidervish, C Gebhardt, N Astman, MJ Gutnick, U Heinemann
Journal of Neuroscience 21 (13), 4600-4608, 2001
Activation of Protein Kinase C Increases Neuronal Excitability by Regulating Persistent Na+ Current in Mouse Neocortical Slices
N Astman, MJ Gutnick, IA Fleidervish
Journal of neurophysiology 80 (3), 1547-1551, 1998
Endogenous polyamines regulate cortical neuronal excitability by blocking voltage-gated Na+ channels
IA Fleidervish, L Libman, E Katz, MJ Gutnick
Proceedings of the National Academy of Sciences 105 (48), 18994-18999, 2008
NMDA receptors in layer 4 spiny stellate cells of the mouse barrel cortex contain the NR2C subunit
AM Binshtok, IA Fleidervish, R Sprengel, MJ Gutnick
Journal of Neuroscience 26 (2), 708-715, 2006
Role of sodium channel subtype in action potential generation by neocortical pyramidal neurons
E Katz, O Stoler, A Scheller, Y Khrapunsky, S Goebbels, F Kirchhoff, ...
Proceedings of the National Academy of Sciences 115 (30), E7184-E7192, 2018
Optogenetic control of mitochondrial metabolism and Ca2+ signaling by mitochondria-targeted opsins
T Tkatch, E Greotti, G Baranauskas, D Pendin, S Roy, LI Nita, ...
Proceedings of the National Academy of Sciences 114 (26), E5167-E5176, 2017
M-current inhibition rapidly induces a unique CK2-dependent plasticity of the axon initial segment
J Lezmy, M Lipinsky, Y Khrapunsky, E Patrich, L Shalom, A Peretz, ...
Proceedings of the National Academy of Sciences 114 (47), E10234-E10243, 2017
Modulation of cardiac sodium channel gating by lysophosphatidylcholine
NA Burnashev, AI Undrovinas, IA Fleidervish, JC Makielski, ...
Journal of molecular and cellular cardiology 23, 23-30, 1991
Ischemic poison lysophosphatidylcholine modifies heart sodium channels gating inducing long-lasting bursts of openings
NA Burnashev, AI Undrovinas, IA Fleidervish, LV Rosenshtraukh
Pflügers Archiv 415, 124-126, 1989
Differential TGF-β signaling in glial subsets underlies IL-6–mediated epileptogenesis in mice
N Levy, DZ Milikovsky, G Baranauskas, E Vinogradov, Y David, M Ketzef, ...
The Journal of Immunology 195 (4), 1713-1722, 2015
The earliest neuronal responses to hypoxia in the neocortical circuit are glutamate-dependent
O Revah, E Lasser-Katz, IA Fleidervish, MJ Gutnick
Neurobiology of disease 95, 158-167, 2016
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